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CD57 / B3GAT1 Monoclonal Mouse Antibody (NK/804)

Anti-CD57 marks a subset of lymphocytes known as natural killer (NK) cells.

Product Attributes

Antibody number

0804

Reactivity (target)

B3GAT1, CD57

Antibody type

Primary

Host species

Mouse

Clonality

Monoclonal

Clone

NK/804

Isotype

IgM, kappa

Molecular weight

~110 kDa

Human gene symbol

B3GAT1

Entrez gene ID

27087

SwissProt

Q9P2W7

Unigene

381050

Immunogen

Recombinant human B3GAT1 protein

Cellular localization

Membrane/cell surface

Species reactivity

Human

Applications

Immunofluorescence, Immunohistology (formalin)

Application notes

Does not react with rat; others not known, Immunohistology formalin-fixed 2-4 ug/mL, Staining of formalin-fixed tissues requires boiling tissue sections in 10 mM Tris with 1 mM EDTA, pH 9.0, for 10-20 min followed by cooling at RT for 20 minutes, Immunofluorescence 0.5-1 ug/mL, Optimal dilution for a specific application should be determined by user

Positive control

Lymph node or Tonsil

Shipping condition

Room temperature

Storage Conditions

Store at 2 to 8 °C, Note: store BSA-free antibodies at -10 to -35 °C

Shelf life

Guaranteed for at least 24 months from date of receipt when stored as recommended

Regulatory status

For research use only (RUO)

Research areas

Cancer, Immunology

Conjugation
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Product Description

Anti-CD57 marks a subset of lymphocytes known as natural killer (NK) cells. Follicular center cell lymphomas often contain many NK cells within the neoplastic follicles. Anti-CD57 also stains neuroendocrine cells and their derived tumors, including carcinoid tumor and medulloblastoma. Anti-CD57 can also be useful in separating type B3 thymoma from thymic carcinoma when combined with a panel that includes antibodies against GLUT1, CD5, and CEA.

This antibody is available purified with BSA/azide at 200 ug/mL, or BSA/azide-free at 1 mg/mL.

References

Okada, T., et al. 1995. Origin of CD57 T cells which increase at tumour sites in patients with colorectal cancer. Clin. Exp. Immunol. 102: 159-166

 

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